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Plasma protein binding is often mentioned as a factor playing a role in pharmacokinetics, pharmacodynamics, and drug interactions

Notably, GPX4-deficient CD4+ and CD8+ T cells fail to expand in the context of acute infections, which is rescued by supplementation of VitE [256]

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NO regulates the AsAGSH cycle via S-nitrosylation of redox enzymes and TFs, increasing redox buffering capacity [58]